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Adenocarcinomas of the lung harbor somatic TP53 gene mutations in a significant proportion of cases. Thus, a rearranged form of mutant p53 protein (mutant p53-24) was originally identified, which is observed particularly frequently in adenocarcinomas. To clarify the significance of mutant p53-24 in adenocarcinoma of the lung, immunohistochemistry was used to detect and semiquantitate it in 33 adenocarcinomas, including 17 cases with TP53 mutations. The results were compared with those of other markers for adenocarcinoma, such as sex hormone receptor (alpha and beta) and carcinoembryonic antigen. In some cases, the ratio of mutant p53-24 to total mutant p53 protein was high, which suggested that it contributed to the malignant conversion of lung adenocarcinoma. However, there was no direct correlation between the presence of mutant p53-24 and sex hormone receptor and carcinoembryonic antigen expression, although it was positively correlated to the presence of TP53 mutations. Furthermore, the ratio of mutant p53-24/total mutant p53 protein was different among the subtypes of adenocarcinoma, and the ratio was higher in patients with TP53 mutations than in those without them. These findings indicate that the presence of mutant p53-24 in adenocarcinoma of the lung depends on TP53 mutations, and that it contributes to malignant progression of adenocarcinoma.Comparison
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